SS-31

• Specifically targets and protects cardiolipin on the inner mitochondrial membrane
• Improves mitochondrial bioenergetics and ATP production
• Reduces mitochondrial reactive oxygen species (ROS) production
• Studied for cardiac protection in heart failure and ischaemia-reperfusion injury
• Investigated for Barth syndrome, age-related mitochondrial decline, and neurodegenerative diseases
• May reverse mitochondrial dysfunction associated with aging

• Preclinical pharmacology studies confirm selective accumulation at the inner mitochondrial membrane via cardiolipin binding
• Animal models show significant improvements in cardiac output, skeletal muscle function, and mitochondrial bioenergetics
• Published in Nature Medicine and Journal of Clinical Investigation: preclinical evidence for cardioprotection and lifespan improvement
• Animal models of ischaemia-reperfusion injury demonstrate reduced mitochondrial ROS and improved post-injury recovery
• Supplied for laboratory and preclinical research purposes only

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